HSV-1
Selected indexed studies
- HSV-1 and Endogenous Retroviruses as Risk Factors in Demyelination. (Int J Mol Sci, 2021) [PMID:34072259]
- Noncanonical autophagy is a new strategy to inhibit HSV-1 through STING1 activation. (Autophagy, 2023) [PMID:37471002]
- The HSV-1 ubiquitin ligase ICP0: Modifying the cellular proteome to promote infection. (Virus Res, 2020) [PMID:32416261]
_Worker-drafted node — pending editorial review._
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Sources
- HSV-1 and Endogenous Retroviruses as Risk Factors in Demyelination. (2021) pubmed
- Noncanonical autophagy is a new strategy to inhibit HSV-1 through STING1 activation. (2023) pubmed
- The HSV-1 ubiquitin ligase ICP0: Modifying the cellular proteome to promote infection. (2020) pubmed
- HSV-1-induced N6-methyladenosine reprogramming via ICP0-mediated suppression of METTL14 potentiates oncolytic activity in glioma. (2024) pubmed
- HSV-1 US3: A Master Mimic Hijacking Host Kinase Networks for Viral Dominance. (2025) pubmed
- HSV-1 employs UL56 to antagonize expression and function of cGAMP channels. (2024) pubmed
- HSV-1 ICP0 dimer domain adopts a novel β-barrel fold. (2024) pubmed
- Genital Herpes Infection: Progress and Problems. (2023) pubmed
- Cellular Processes Induced by HSV-1 Infections in Vestibular Neuritis. (2023) pubmed
- State of the Art: Acute Encephalitis. (2023) pubmed