ER stress PERK
Selected indexed studies
- PERK-ATAD3A interaction provides a subcellular safe haven for protein synthesis during ER stress. (Science, 2024) [PMID:39116259]
- ER and Nutrient Stress Promote Assembly of Respiratory Chain Supercomplexes through the PERK-eIF2α Axis. (Mol Cell, 2019) [PMID:31023583]
- UPR proteins IRE1 and PERK switch BiP from chaperone to ER stress sensor. (Nat Struct Mol Biol, 2019) [PMID:31695187]
_Worker-drafted node — pending editorial review._
Connections
No connections recorded yet.
Sources
- The unfolded protein response: controlling cell fate decisions under ER stress and beyond. (2012) pubmed
- PERK-ATAD3A interaction provides a subcellular safe haven for protein synthesis during ER stress. (2024) pubmed
- ER and Nutrient Stress Promote Assembly of Respiratory Chain Supercomplexes through the PERK-eIF2α Axis. (2019) pubmed
- UPR proteins IRE1 and PERK switch BiP from chaperone to ER stress sensor. (2019) pubmed
- Liver-specific gene PGRMC1 blocks c-Myc-induced hepatocarcinogenesis through ER stress-independent PERK activation. (2025) pubmed
- The Role of the PERK/eIF2α/ATF4/CHOP Signaling Pathway in Tumor Progression During Endoplasmic Reticulum Stress. (2016) pubmed
- ER stress sensor PERK drives ferroptosis by disrupting MAMs-mediated mitochondrial homeostasis and promoting mtROS accumulation in acute myocardial infarction. (2025) pubmed
- Inhibition of vascular smooth muscle cell PERK/ATF4 ER stress signaling protects against abdominal aortic aneurysms. (2025) pubmed
- ER stress tolerance is regulated by copper-dependent PERK kinase activity. (2025) pubmed
- ER stress sensor PERK promotes T cell pathogenicity in GVHD by regulating ER-associated degradation. (2025) pubmed