chronic PM2.5
Selected indexed studies
- The Effects and Pathogenesis of PM2.5 and Its Components on Chronic Obstructive Pulmonary Disease. (Int J Chron Obstruct Pulmon Dis, 2023) [PMID:37056681]
- Impaired AT2 to AT1 cell transition in PM2.5-induced mouse model of chronic obstructive pulmonary disease. (Respir Res, 2022) [PMID:35337337]
- PM2.5 increases susceptibility to acute exacerbation of COPD via NOX4/Nrf2 redox imbalance-mediated mitophagy. (Redox Biol, 2023) [PMID:36608590]
_Worker-drafted node — pending editorial review._
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Sources
- Chronic Exposure to Bioaerosols in PM2.5 from Garbage Stations Accelerates Vascular Aging via the NF-κB/NLRP3 Pathway. (2024) pubmed
- PM2.5 increases susceptibility to acute exacerbation of COPD via NOX4/Nrf2 redox imbalance-mediated mitophagy. (2023) pubmed
- Impaired AT2 to AT1 cell transition in PM2.5-induced mouse model of chronic obstructive pulmonary disease. (2022) pubmed
- Ovarian Dysfunction Induced by Chronic Whole-Body PM2.5 Exposure. (2020) pubmed
- Clinicopathological relationship between PM2.5 exposure and acute exacerbations of chronic obstructive pulmonary disease. (2025) pubmed
- PM2.5 induces ferroptosis in chronic obstructive pulmonary diseases via the GSK-3β/NRF2 pathway. (2025) pubmed
- The Effects and Pathogenesis of PM2.5 and Its Components on Chronic Obstructive Pulmonary Disease. (2023) pubmed
- Selenomethionine mitigate PM2.5-induced cellular senescence in the lung via attenuating inflammatory response mediated by cGAS/STING/NF-κB pathway. (2022) pubmed
- Function of PM2.5 in the pathogenesis of lung cancer and chronic airway inflammatory diseases. (2018) pubmed
- Melatonin ameliorates PM2.5-induced airway inflammation and apoptosis by PERK/eIF2α/ATF4/CHOP in chronic obstructive pulmonary disease mice. (2025) pubmed